Changes

The abstracts of all chapters of [[Nidana Sthana]] are presented here.  

<p style='text-align:justify;'>The abstracts of all eight chapters of [[Nidana Sthana]] are presented here for introductory knowledge about its chapters. For more knowledge, the readers can go to the individual chapters. </p>

== Chapter 1. Chapter on Fundamental Principles of Diagnosis and Fever – [[Jwara Nidana]] ==  

== Chapter 1. Chapter on Fundamental Principles of Diagnosis and Fever – [[Jwara Nidana]] ==  

<div style="text-align:justify;">The third chapter describes the etiology, pathogenesis, clinical features and therapeutics of lumps in abdomen. Gulma, a (stable or transitory) growth that could occur anywhere in the body is primarily caused by a vitiated vata. The description given in the text cannot be correlated with any single entity of conventional medicine, but denotes localized intra-abdominal swellings of multiple origin in terms of their cause, site, features etc. In patients weakened by or recuperating from diseases or cleansing therapies (vamana, etc.), or suffering from very stressful conditions, vata gets vitiated and enters the mahasrotas (gastrointestinal tract) causing the formation of gulma. Amongst the five types of gulma, sannipatika gulma is incurable whereas remaining can be managed according to dosha involvement. </div>

<div style="text-align:justify;">The third chapter describes the etiology, pathogenesis, clinical features and therapeutics of lumps in abdomen. Gulma, a (stable or transitory) growth that could occur anywhere in the body is primarily caused by a vitiated vata. The description given in the text cannot be correlated with any single entity of conventional medicine, but denotes localized intra-abdominal swellings of multiple origin in terms of their cause, site, features etc. In patients weakened by or recuperating from diseases or cleansing therapies (vamana, etc.), or suffering from very stressful conditions, vata gets vitiated and enters the mahasrotas (gastrointestinal tract) causing the formation of gulma. Amongst the five types of gulma, sannipatika gulma is incurable whereas remaining can be managed according to dosha involvement. </div>

== Nidana Sthana Chapter 4. Diagnosis and etiopatheogenesis of Obstinate Urinary Disorders including diabetes – [[Prameha Nidana]] ==

== Chapter 4. Diagnosis and etiopatheogenesis of Obstinate Urinary Disorders including diabetes – [[Prameha Nidana]] ==

<div style="text-align:justify;">This chapter describes the disease prameha (group of obstinate urinary disorders including diabetes) as well as its etiopathogenesis, types, clinical features, prognosis, prodromal symptoms, complications, and a line of treatment. Though it is predominantly a kapha dosha disorder, there are variants involving all three doshas and ten dushya (body tissues and other elements). Excessive and  turbid urination are the cardinal signs of all prameha. All those urinary and systemic diseases that cause copious quantities, as well as the abnormal (including turbid) quality of urination, come under the umbrella of prameha. This chapter briefly explains the importance of interaction of exogenous factors, endogenous and tissue response factors in the manifestation of the disease. The concept of genetic susceptibility to disease and effect of chronic disease on genes is also incorporated in this chapter. </div>

<div style="text-align:justify;">This chapter describes the disease prameha (group of obstinate urinary disorders including diabetes) as well as its etiopathogenesis, types, clinical features, prognosis, prodromal symptoms, complications, and a line of treatment. Though it is predominantly a kapha dosha disorder, there are variants involving all three doshas and ten dushya (body tissues and other elements). Excessive and  turbid urination are the cardinal signs of all prameha. All those urinary and systemic diseases that cause copious quantities, as well as the abnormal (including turbid) quality of urination, come under the umbrella of prameha. This chapter briefly explains the importance of interaction of exogenous factors, endogenous and tissue response factors in the manifestation of the disease. The concept of genetic susceptibility to disease and effect of chronic disease on genes is also incorporated in this chapter. </div>